I find the concept of taking air breaks to manage oxygen toxicity while decompressing comparable to using a paper towel to mop up an incoming tide at the beach. Or put another way, air breaks in this context are about as useful as ashtrays on a motorcycle.
Allow me to explain. I believe oxygen toxicity is one of the biggest risks to recreational divers, especially technical divers, but air-breaks as commonly described and executed, are no substitute for proper CNS planning… and are useless as a CNS management tool in any event.
Oxygen toxicity is a condition resulting from the harmful effects of breathing oxygen at elevated partial pressures. The most serious form of oxygen toxicity has the potential to affect a diver’s central nervous system and is a result of breathing very high-partial pressures (more than one bar or atmosphere) for a relatively short period of time (less than a few minutes at extreme levels). This type of toxicity may result in a clonic-tonic seizure; which in the water usually means death by embolism or drowning. Historically, this central nervous system condition was called the Paul Bert effect. The less problematic whole-body or pulmonary condition – a function of breathing lower partial pressures (less than one bar) over much longer periods – goes under the name the Lorrain Smith Effect, after the researchers who pioneered its discovery and description in the late 19th century.
I have heard and read that divers manage both Paul Bert and even Lorrain Smith effects by taking a short “air-break” during moderately long decompressions. The typical scenario is this: A diver conducts a deep or deepish dive which earns her a lengthy series of staged decompression stops on her way back to the surface. She finishes her dive by breathing pure oxygen at 6 metres on up. In this scenario, the decompression schedule requires the diver to breathe oxygen for around 20 minutes. There are a pile of variations on this theme, but the common thread is a fair amount of time breathing a gas that is delivering around 1.6 bar of oxygen… by the way, the NOAA limit for exposure to 1.6 bar of oxygen for a diver is 45 minutes, so this type of exposure does load a diver with the potential for a CNS incident… there is no argument there.
The “air-break” myth goes something like this. At some point during her spell breathing pure oxygen – sometimes at the end and sometime mid-stream – the diver will “RESET” her CNS “clock” by switching from breathing oxygen to breathing bottom mix, air, a less oxygen-rich nitrox (typically the mix she was breathing during her ascent to her final stops). Let’s illustrate the air-break protocol with a dive profile calling for a final decompression stop for 21 minutes at six metres or 20 feet. In this example, the diver might use oxygen for ten minutes, and then switch to say an EAN50 for five minutes, and finally switch back to oxygen for eleven minutes to finish up their deco. Typically, as in this example, the time spent on an “air-break” is not credited against the decompression obligation.
What I have yet to hear fully explained is how a five-minute break from breathing pure O2 resets a diver’s CNS loading during this procedure. Actually, you may also read postings from divers who rely on the same technique to manage Lorrain Smith effect, which shows an even greater misinterpretation of the mechanism behind the syndrome*.
OK, let’s take a step back and turn on the logic filter. According to NOAA – the folks who literally set the standards for nitrox use in the recreational dive community – a period of 20 minutes breathing oxygen at 6 metres – a practice that delivers a partial pressure or oxygen depth of around 1.6 bar/ata – has a corresponding time limit of 45 minutes. When we calculate the CNS loading for a dive, we are taught to account for the CNS loading for ALL phases of the dive. That’s to say, every minute spent breathing elevated levels of oxygen. Let’s ignore whatever came before during our example dive, and let us just focus on what happens at six metres or 20 feet. In a nutshell: The diver has to account for 20 minutes on pure oxygen. The NOAA tables don’t give a rat’s behind whether those 20 minutes are accumulated in one lump or two… or three or four. Twenty minutes is 20 minutes and uses up about 44-45 percent of the total allowable time regardless! The five minutes breathing another gas – in our example we can say she used EAN50 delivering an oxygen partial pressure of about 0.8 bar – simply adds a little to the total CNS loading, albeit a very tiny about (less than one percent). There is nothing in the NOAA dive manual or any of Hamilton’s published work that tells us anything different.
Now, to set the record straight, faced with the situation outlined above and breathing pure oxygen for that long, the chances are that I would take an air-break and recommend taking one to my team-mates; however, it has NOTHING to do with CNS but rather to help optimize off-gassing.
Oxygen is a vasoconstrictor – it causes some blood vessels to shut or partially shut – which may have some effect on general perfusion levels. This does not seem like a great plan for those of us trying to eliminate dissolved inert gas.
The bottom line is this: Let’s agree to take a break from pure O2 during our deco, but let’s not confuse the issue by suggesting that doing so magically helps manage CNS toxicity. Better yet, let’s opt to employ a better option and a slightly more helpful gas. But more about that later.
* Prolonged breathing of gas with an Fio2 (Fractional Inspired Oxygen) greater than 60 kPa (0.6 bar/ata)can lead to pulmonary toxicity and eventually irreversible pulmonary fibrosis, but this takes many hours or days and does not constitute an issue for the rank and file technical diver. Most likely, the “burning” sensation and pulmonary toxicity like symptoms mentioned by technical divers breathing oxygen and oxygen-rich gas during recreational decompression is a function of breathing cold, dry air (the dew-point of oxygen in the cylinders in my fill station is marked as -40! That’s dry.) This air has the ability to dry the mucus membranes lining our lungs and bringing on something called dry-air asthma. A less far-fetched probable outcome than pulmonary toxicity.
Kathy Dowsett
www.kirkscubagear.com
